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高壓氧治療大幅減輕糖尿病足部潰瘍及其併發症: 糖尿病一直高居先進國家十大死亡疾病之列, 糖尿病的併發症有:腦中風、冠狀動脈狹窄、眼底血管病變、腎功能衰竭與足部缺血性潰瘍等   高壓氧治療問題傷口的機轉: 1.提升組織含氧量,解除缺氧和水腫引起之惡性循環。 2.因此改善血液循環,纖維母細胞有足夠的營養和氧氣產生大量膠原蛋白,加速傷口之癒合。 3.促進原生幹細胞由骨髓轉移到缺氧的部位,提升血管和組織再生的能力。 4.供應吞噬性白血球進行殺菌時所需15 倍的耗氧量,製造大量過氧游離基,執行殺菌的功能。 5.足夠的氧氣可斷絕厭氧性細菌滋長的機會,快速裂解其能溶解肌肉損傷心臟血管及神經系統等之二十餘種毒素。 6.幫助抗生素到達感染的患部,有效執行殺菌的工作。 7.增加惰性細菌體內氧化還原之活性,提升抗生素殺菌時之著力點。 8.抑制有害的發炎反應,降低細菌分泌內毒素引起之敗血性休克和多重器官的衰竭。 https://www.houstonmethodist.org/newsroom/houston-methodist-sugar-land-hospital-offers-advanced-treatment-for-nonhealing-wounds/   HOUSTON METHODIST SUGAR LAND HOSPITAL OFFERS ADVANCED TREATMENT FOR NONHEALING WOUNDS SUGAR LAND - July 2, 2018 Media Contact:Kaelyn Bujnoch, 281.274.8085   For people living with diabetes, even the smallest scrape can lead to potentially dangerous circumstances. High blood sugar can damage nerves, which may cause numbness in the feet and legs, and lead to a narrowing and hardening of blood vessels, which can result in less oxygen being circulated throughout the body to aid in healing.   If you have a wound that doesn’t heal properly, early treatment can help you get back on your feet. The Houston Methodist Wound Care Program at Sugar Land uses a multidisciplinary approach for treating wounds that won’t heal.   DIABETIC FOOT WOUNDS   Timothy Oppermann, M.D., board-certified general surgeon with Houston Methodist Surgical Associates at Sugar Land, encourages patients to take a proactive approach.   “Diabetes accounts for about 60 percent of lower limb amputations not related to injury in adults, clearly illustrating the importance for diabetic patients to closely monitor their health and take quick action to provide the proper care to even the smallest wound, particularly in the feet,” Oppermann said.   Warning signs of a chronic, nonhealing wound include:   Persistent pain in the area around the wound Discoloration of the wound’s edges Increased drainage from the wound site Redness or swelling around or spreading away from the wound   HYPERBARIC OXYGEN THERAPY   The program’s doctors and nurses are certified in wound care medicine, and work with patients and their primary care physician to monitor, manage and treat wounds with the most advanced treatments available, including compression wraps, antimicrobial dressings, bioengineered skin substitutes and hyperbaric oxygen therapy (HBOT).   Michael Yaakovian, M.D., board-certified general surgeon with Houston Methodist Surgical Associates at Sugar Land, treats patients with diabetic foot problems and other nonhealing wounds. “Hyperbaric therapy supplies oxygen under pressure that supersaturates the red blood cells and plasma with oxygen, so when blood is supplied to the area of the wound, these supercharged blood cells help repair the wound and kill the infection, leading to the generation of new blood vessels and tissue growth,” Yaakovian explained.   FIVE SELF-CARE TIPS   For people with diabetes, managing wounds to prevent complications requires care and diligence. Take good care of your feet with the following tips.   Inspect and wash feet daily. With poor sensation, wounds could go unnoticed. Keep wounds clean and dry to reduce risk of infection. File your toe nails. Clipping may increase the risk for skin injury. Wear proper-fitting shoes and socks at all times. Control your blood sugar, blood pressure and cholesterol to slow down the progression of diabetic complications.   SEE A WOUND SPECIALIST   If a wound does not show signs of healing, it should be evaluated by a wound specialist. To schedule an appointment with Timothy Oppermann, M.D. or Michael Yaakovian, M.D., call 281.275.0860. Visit houstonmethodist.org/wound-sl to learn more about our services.   Visit our Facebook page at fb.com/methodistsugarland for the latest news, events and information.  more
在一次罕見的中風使她的視力幾乎喪失後,一位藝術家重新獲得了視野。高壓氧療法是關鍵。 Legally blind Northstate woman regains her vision through oxygen therapy   by Julia Avery Saturday, March 7th 2020 AA                                             02:13 02:16               <p>{/p} REDDING, Calif. — A Northstate artist regained her vision after nearly lost her eyesight from a rare stroke. Hyperbaric Oxygen Therapy was the key. Dr. Julie Hamilton with Mercy Medical Center's Wound, Healing, and Hyperbaric Center said the therapy works, but it is time-sensitive. “So Darcy came in late Thursday afternoon. Dr. Bowen had called us and said, ‘I have a patient that needs your help, there’s no other option and I’m really worried. She’s got a Central Retinal Artery Occlusion and like any kind of stroke, which is basically what that diagnosis is, just minutes and seconds matter,” said Hamilton. A third of Darcy Wilson's vision was blocked in her right eye. Her Ophthalmologist sent her to Mercy to try the hyperbaric chamber. "The reason hyperbaric oxygen therapy works is what you’re doing, is trying to drive oxygen to a part of the body where there isn’t any," said Hamilton. "If you do that overtime at 100 percent concentration and the added pressure, your body actually forms new blood vessels” Wilson said she was scared to get into the chamber at first, but she's an artist and losing her vision isn't an option. “Doing paintings, painting with my grandchildren is so important and such an important part of my life," said Wilson. She lives in Weaverville, but was willing to make the drive and get over her fears of the chamber. “Obviously in a situation like this there’s no question, no matter what her fears were, she was going to go in that chamber.” After nine 90 minute treatments, she regained her vision. “I just can’t say enough how grateful I am to all of them for helping me get my vision back," said Wilson. "It was amazing going through the treatment, terrifying but amazing, amazing their dedication to their work”     more
加拿大Judy Dan使用範美高壓氧艙 https://ontariowoundcare.com/treatment/hyperbaric-medicine/?fbclid=IwAR2w8zUP7plDjR9mI6PuNWrhT1WBNsfSJSzEYwKFgx4F3XlelIwQFaTB_4A Save a Leg SAVE A LIFE. Hyperbaric Oxygen Therapy (HBOT): How it works Hyperbaric Oxygen Therapy (HBOT) involves administering 100% oxygen to patients in a specially constructed chamber. The atmospheric pressure is increased two to three times normal for 90 minutes per session. This has the effect of: Increasing vastly the oxygen concentration of the patient’s blood. Saturating the wound with oxygen. Enabling cells to function that fight infection and repair wounds.   more
https://www.youtube.com/watch?v=78PXByMI5J8&t=12s more
https://hbot.com/the-secret-to-growing-new-brain-cells-sleep-exercise-and-hyperbaric-oxygen-by-veronica-fern-mcelarney-stanford-university/   The Secret to Growing New Brain Cells: Sleep, Exercise and Hyperbaric Oxygen?By Veronica Fern-McElarney, Stanford University  September 15, 2012 Category: News, Press No Comments A 33-year-old drunk driver wraps his pick-up truck around a tree and is brought to the emergency room at a small, community hospital in Slidell, Louisiana. His emergency room doctor, Dr. Paul Harch, recalled the scene. “You know, high-speed, straight into a pick-up, no seatbelt, and the flexion injury rendered him paralyzed immediately. By the time they got him off the floorboard of the truck…he had a flicker of movement in his one big toe; within forty minutes he was densely paraplegic. At three in the morning the neurosurgeon, radiologist and myself looked at each other…and the only explanation was that he had a vascular injury to his spinal cord. And almost in unison we said, “Gosh, I wonder what he could do with a little oxygen?” I put him in the hyperbaric chamber and he moved his toe. When we took him out, he had sensation down in the foot. Incrementally, every time I put him in, he got more and more sensation. In seventeen days, he walked out of the hospital. That just blew everybody away.” Harch, a physician at the Louisiana State University Medical Center and Inaugural (past) President of the International Hyperbaric Medicine Association, has been treating patients in hyperbarics since 1985. Patients are dosed with high-pressure oxygen by placing them in a decompression chamber, a treatment usually associated with scuba divers with decompression sickness. Harch, 53, who trained at Johns Hopkins University had “some very high aspirations.” Unfortunately, after starting his internship in general surgery, he was hit by a car and badly injured in the accident, derailing his career plans. He ended up doing emergency medicine in Slidell, 90 miles from the Gulf of Mexico. Disappointed with the direction his career was taking, he nearly quit medicine. He gave notice and told his colleagues he was looking to make his mark elsewhere. But after he “decided to quit my pissing and moaning,” he realized that he was intrigued by decompression illness, and curious about the fact that there was no literature on diver neurology. “Everybody thinks of decompression sickness as the bends –joint pain, bent over, and so on. In fact, three-quarters to four-fifths of all decompression sickness is neurological—mostly spinal cord, but a good proportion of that is brain-based. Eight and a half decades of diving medicine and nobody could answer some simple questions about what was going on in the brain…an opportunity was being presented to me that I couldn’t walk away from – to change and revolutionize the treatment of brain injury.” Until recently, most scientists believed that the brain contained a finite number of cells, depleted over the course of human life. However, recent research has overturned that idea. The most important findings have implied that two factors are required for brain cell growth – not crossword puzzles and not advanced math, thankfully – but sufficient sleep and exercise. Dr. Astrid Bjørnebekk found that exercise stimulates the production of new brain cells (reported in ScienceDaily, June 29, 2007). Bjørnebekk’s study aimed to decrease depression through exercise, which increases blood flow to the brain. Scientists believe this sea change signals future relief of human suffering for a variety of “incurable” neurological ailments – Stroke, Parkinson’s disease, Alzheimer’s, and chronic Traumatic Brain Injury (TBI). So how did Harch reach his seemingly zany conclusion? In the late 80’s, Harch realized divers were coming to him with symptoms of decompression sickness long after diving, and no longer had the nitrogen bubbles in their systems that cause decompression illness. Further study led him to believe that what he was observing was low blood-flow brain injury. Harch conducted experiments using lower-pressure hyperbaric oxygen treatments (HBOT) over longer time spans. Soon Harch was using the new protocol to treat a variety of neurological problems, including chronic TBI, Cerebral Palsy, Autism, and Stroke. Time after time, Harch saw improvement in the cognitive function of his patients and in brain blood flow scans (SPECT images) of patients’ brains. Harch put his clinical procedure under an experimental protocol for six years: a SPECT scan, one hyperbaric treatment, followed by another scan, to compare for improvement in brain blood flow. Then he treated people in blocks of forty treatments, to assess response, and indexed it to imaging and other clinical indicators. He found that the imaging predicted who would improve. After a while, he didn’t even need imaging because so many people responded to the treatment; it was reproducible. As Harch began to present his research, he realized that scientists didn’t want to believe his findings “because they violated 100 years of neurology.” Seeking to silence his critics, Harch embarked on a series of animal studies. His funding was minimal and came in the form of a $50,000 gift from Louis Ross, a patient’s husband who owned a dairy. In the first twelve rats studied, the team had statistically significant results, but the physiologist who designed the research model refused to publish it, believing the data was “flukey.” Harch’s team repeated the study with thirty rats, but a malfunction of the lab equipment nixed the results. They then planned a study using five times the original number of rats. This study coincided with a heat wave in New Orleans in July 1998. The rats were shipped in an un-air-conditioned van and expired before they reached the lab. Finally, in 2001, the team’s animal research generated very powerful results, even more so than the previous pilot trial. “But they sat on it for a couple of years trying to find what we had done wrong—-it was like the O.J. trial, you know?…Here we had all these positive results, but they just knew something had to be wrong with the methodology. It was a fifteen-year-old model when we started in 1994. It finally got published and that was last year (2007).” Finally, after 17 years, the findings were published in Brain Research.[i] They describe a ground-breaking improvement of chronic brain injury in animals. Results were impressive, and reconfirmed their treatment outcomes with human patients. The researchers measured the vascular density of the rats’ brains where the injury occurred, and correlated this with the rats’ cognitive performance. In comparison to the two control groups, the group treated with HBOT showed markedly improved vascular density in the injured area, which was associated with improved cognitive performance. In other words, Harch and his colleagues found that HBOT significantly increased blood volume and flow to the affected brain areas and that helped the rats regain some of their lost function. Dr. Gary K. Steinberg, Chair of the Department of Neurosurgery at Stanford University School of Medicine warns, “I would be cautious about drawing any definitive conclusions regarding the efficacy of hyperbaric oxygen for treating neurologic disease, including TBI. The preclinical (animal) results are equivocal and the clinical reports only anecdotal. Although the treatment appears safe, further controlled studies in patients are required before it can be recommended as beneficial therapy.” Insurgents drove a truck laden with 2,000 pounds of explosives into the building where the Marines were holed-up in the middle of Ramadi. Most of the guys in the detachment were knocked out by the massive explosion. In no time, they were under attack and outnumbered by seventy-eight insurgents to their thirty-three – needing help, some of the soldiers were shaking a young man to rouse him from his unconscious state. He regained consciousness, sat up, and they were hit with the second explosion – a rocket-propelled grenade against the wall. The Marine lost consciousness for the second time in fifteen seconds. “Whenever there’s an explosion like that, on the opposite side of the wall, everybody’s knocked out. It’s similar to what happens with concussive explosions and hollow viscouses in our body, lungs and intestines; the shock force does greater damage at the air-tissue interface. So he wakes up and he’s in the middle of a fire-fight…a minimum of seven hours or so… cerebral dysfunction is not appreciated because you’re in a war zone—it’s heightened awareness, poor sleep…hyper-vigilance. It’s not until they get home that…the effects of the brain injury become manifest.” Apart from giving drunk drivers a second chance, Harch’s findings have important implications for Iraq War veterans. As many as 400,000 veterans have been exposed to concussive force and possibly suffered a TBI. According to one patient—a Marine—every man in his thousand-man battalion has been exposed to IED’s and a minimum of 50% have been knocked unconscious. Harch: “It’s considered a non-injury. You’re a wimp if you have symptoms. Unless you’re bleeding or have had an appendage blown away, or lost an eye, you’re not considered injured.” TBI affects over 1.4 million Americans annually. The leading cause is falls, followed by motor vehicle crashes.[ii] Moreover, blasts are a leading cause for active duty military personnel. Called the “signature injury” of the Iraq War, some accounts estimate that up to 60% of injuries related to roadside bombs in Iraq are TBIs. Symptoms include memory loss and communication problems; even worse, perhaps, are the behavioral and psychological ramifications. Formerly balanced, responsible and mature adults may become impulsive, irrational, and depressed. Harch recounted several stories about patients who had become psychologically dysfunctional, “demented,” “suicidal,” “violent” and “a throw-away.” Some patients were institutionalized, diagnosed as untreatable. One commercial diver was refused medical coverage by the company doctor who ruled that his injury as a result of diving was inconclusive. The patient loaded two pistols and went to the company headquarters in New Orleans. Luckily, he called his brother to say good-bye; his brother intervened and put him into treatment with Harch. The patient is now functional and Harch was able to prove his medical case to the company with SPECT imaging. The U.S. military command does not appear to be interested in HBOT. Harch and a colleague submitted separate grant proposals to the military and were turned down. Harch said, “We went to the top guy in the Army who oversees the brain injury program…and it was a very depressing yet enlightening discussion. It went nowhere…it was surprising, the response we got.” Brigadier General Patt Maney was told he is the highest ranking soldier wounded in Afghanistan by a roadside bomb to survive a TBI. A state court judge in Florida, Maney was called up to service in his capacity as an Army Reservist. Maney said that in addition to TBI, soldiers frequently suffer other physical injuries, like wounds from shrapnel, loss of limbs, burns, and broken teeth –“polytrauma.” Maney was treated one year after his injury using the Harch-prescribed HBOT protocol at George Washington University Hospital. Describing his experience, Maney said, “The treatments are easy if one doesn’t have a problem with claustrophobia. Some hyperbaric facilities have multiple person chambers but GW uses one-person chambers. The chambers are clear cylinders with closed ends. A patient is slid into the tube and the end is closed. A technician or physician stays outside the chamber but with the patient the entire time. Initially, I watched television during the [one-hour] dives but as I got more accustomed to the dives, I frequently napped.” Maney said that his wife noticed improvements in his cognition following 8-12 dives, while he observed improvements after 12-14 dives. After 20 dives, his friends commented that he seemed more socially engaged. “From my experience, I believe that HBOT should be the foundational treatment for all soldiers with a TBI. It’s really disappointing that some military medical practitioners decline to recognize the documented scientific advances, possibilities and results of HBOT. The TBI wounded and their families need effective treatment now.” A 2006 study (Finkelstein et al) states that costs related to TBI totaled $60 billion in the year 2000 – actual medical costs as well as lost productivity. This includes over 5.3 million TBI survivors who need ongoing help to carry out the daily activities of living. One of the major advantages of HBOT is its relatively low cost; 40 sessions of HBOT cost about $4,000. Compare this to the financial and human costs of institutionalizing a young person for 40 years. In 2002, Harch testified to Congress about the costs of HBOT relative to taking care of the untreated. “Forty percent of my practice is neurologically injured children…who cost on average, 2.1 times as much to educate as a non-injured child. There are 6.548 million…children in the nation…[which cost taxpayers]…a total of $55.7 billion. For many of these children, if they had been treated immediately upon injury, the costs drop to often less than $1,000.”[iii] Of the many who suffer a TBI annually, 50,000 die, 235,000 are hospitalized, and 1.1 million are treated and released from hospital emergency rooms. The typical person affected is young and male. One patient who fits the profile is 17-year-old Curt Allen, Jr., injured in a 2004 automobile accident. Video details the young man’s presentation following rehabilitation in “the best residential facility in the state of Louisiana,” from which he was dismissed for failure to progress. As he receives HBOT, Allen’s condition improves from practically non-responsive to walking, joking and talking. Harch is currently involved in a ten-year stroke study while he continues to pursue funding to conduct human trials for chronic TBI. “The most important thing to know is that this is a generic drug for growth and repair of brain injury—both acute and chronic. Don’t wait for your doctors to come around to understanding this. It’s a low-risk treatment. Go out and seek a physician at a facility who will do this. Get this treatment for you or your loved one.”   more
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